Analytical Data
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基因名
ACAD9
- Application
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别名
ACAD9Complex I assembly factor ACAD9; mitochondrial; Acyl-CoA dehydrogenase family member 9
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种属
Human
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表达系统
E. coli
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标签
GST-tag at N-terminal.
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纯度
Greater than 90% as determined by SDS-PAGE.
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蛋白编号
Q9H845
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表达区间
1- 621a.a
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氨基酸序列
MSGCGLFLRTTAAARACRGLVVSTANRRLLRTSPPVRAFAKELFLGKIKKKEVFPFPEVSQDELNEINQFLGPVEKFFTEEVDSRKIDQEGKIPDETLEKLKSLGLFGLQVPEEYGGLGFSNTMYSRLGEIISMDGSITVTLAAHQAIGLKGIILAGTEEQKAKYLPKLASGEHIAAFCLTEPASGSDAASIRSRATLSEDKKHYILNGSKVWITNGGLANIFTVFAKTEVVDSDGSVKDKITAFIVERDFGGVTNGKPEDKLGIRGSNTCEVHFENTKIPVENILGEVGDGFKVAMNILNSGRFSMGSVVAGLLKRLIEMTAEYACTRKQFNKRLSEFGLIQEKFALMAQKAYVMESMTYLTAGMLDQPGFPDCSIEAAMVKVFSSEAAWQCVSEALQILGGLGYTRDYPYERILRDTRILLIFEGTNEILRMYIALTGLQHAGRILTTRIHELKQAKVSTVMDTVGRRLRDSLGRTVDLGLTGNHGVVHPSLADSANKFEENTYCFGRTVETLLLRFGKTIMEEQLVLKRVANILINLYGMTAVLSRASRSIRIGLRNHDHEVLLANTFCVEAYLQNLFSLSQLDKYAPENLDEQIKKVSQQILEKRAYICAHPLDRTC
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分子量
95.2 KDa
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内毒素
< 1.0 EU per μg protein as determined by the LAL method.
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性状
Freeze-dried powder
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缓冲液
PBS, pH7.4, containing 0.01% SKL, 1mM DTT, 5% Trehalose and Proclin300.
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复溶方法
Reconstitute in ddH2O to a concentration of 0.1-0.5 mg/mL. Do not vortex.
- 个性化定制
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稳定性测试
The thermal stability is described by the loss rate. The loss rate was determined by accelerated thermal degradation test, that is, incubate the protein at 37℃ for 48h, and no obvious degradation and precipitation were observed. The loss rate isless than 8% within the expiration date under appropriate storage condition.
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保存条件 & 期限
Samples are stable for up to twelve months from date of receipt at -20℃ to -80℃. Store it under sterile conditions at -20℃ to -80℃. It is recommended that the protein be aliquoted for optimal storage. Avoid repeated freeze-thaw cycles.
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运输条件
In general, recombinant proteins are supplied as lyophilized powder and shipped at ambient temperature. For bulk packages, the proteins are provided as frozen liquid and shipped with blue ice, unless otherwise requested by the customer.
Quality inspection process
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Protein Description
ACAD9, also known as acyl-CoA dehydrogenase family member 9, plays a crucial role in the mitochondrial fatty acid oxidation pathway, particularly in the metabolism of medium-chain fatty acids. Research has highlighted its importance in maintaining energy homeostasis and its involvement in various metabolic disorders. Mutations or deficiencies in ACAD9 are linked to complex genetic conditions, including severe infantile metabolic crises, suggesting its vital function in cellular energy production. The study of ACAD9 recombinant proteins has gained momentum as scientists strive to understand its structure-function relationship, regulation, and interactions with other metabolic enzymes. Additionally, investigating ACAD9 could reveal insights into potential therapeutic strategies for related metabolic diseases. Due to the limited availability of natural ACAD9, producing recombinant forms enables the exploration of its biochemical properties and interactions, facilitating drug design and the development of targeted therapies for mitochondrial dysfunction. Overall, ACAD9 remains a significant focus within metabolic research, aiming to unravel the complexities of fatty acid metabolism and its implications for human health.












